A case of Potter syndrome due to posterior urethral valve.

is widely accepted that for lung development to proceed normally, physical space in the fetal thorax must be adequate and amniotic fluid must be brought into the lung with fetal breathing movements. Thus, it follows that extrinsic compression of the fetal thorax causes hypoplasia, either by inhibiting respiratory movements or by forcing out fluid from the lungs. The latter hypothesis was supported by Nicolini et al, on the basis of an observed reduction in amniotic pressure. Hence, the instillation of artificial amniotic fluid could restore abnormal amniotic pressure and help prevent pulmonary hypoplasia. The combination of amnio-infusion and bladder aspirations has achieved a successful outcome in the management of obstructive foetal hydronephrosis.